What condition may result from neutrophil aggregation during cardiopulmonary bypass (CPB)?

Neutrophil aggregation during cardiopulmonary bypass (CPB) can lead to complement activation, which is an important aspect of the immune response. During CPB, neutrophils can become activated due to the contact with foreign surfaces, leading to the release of inflammatory mediators. This activation can trigger the complement system, a series of proteins that play a key role in the immune response.

When complement activation occurs, it can contribute to both local and systemic inflammatory responses, which may lead to complications such as organ dysfunction or damage. This connection highlights the significance of managing the inflammatory response during CPB to minimize the impact on patient outcomes.

The other choices do not directly correlate with the effects of neutrophil aggregation. For instance, while decreased circulation may be a consequence of various factors during CPB, it is not a primary result of neutrophil aggregation. Increased oxygenation and reduced anesthetic effects are also unrelated outcomes of neutrophil aggregation during the procedure. Hence, the association between neutrophil aggregation and complement activation is crucial for understanding the inflammatory processes that can occur during cardiopulmonary bypass.